Nature Cell Biology, Published online: 14 August 2026; doi:10.1038/s41556-026-02048-x Kim et al. demonstrate a non-canonical function for EZH2 in modulating cholesterol metabolism and tumour development by interacting with SREBP2, which may be targeted by PROTACs to disrupt its oncogenic activity.
An EZH2–SREBP2 axis promotes cholesterol biosynthesis and represents a noncanonical vulnerability in tumorigenesis
Gang Greg Wang

