[Biochimica Et Biophysica Acta-Molecular Basis of Disease] Investigators identified the chromatin remodeler HELLS as a critical oncogenic driver in prostate cancer and elucidated a previously unrecognized post-translational cascade that governs its protein stability.
Neddylation-Triggered Ubiquitination and Proteasomal Degradation of HELLS Inhibits Prostate Cancer Growth via Genomic Instability
Emily Salmini


