Epistatic chromatin remodeling during barley response to powdery mildew by ATAC-Seq

Abstract Understanding the molecular basis of plant-pathogen interactions is critical for advancing crop protection strategies. Powdery mildew, caused by the obligate fungal pathogen Blumeria hordei ( Bh ), is a threat to barley production worldwide. We exploited time-course ATAC-Seq of barley and derived immune mutants infected with Bh to infer chromatin accessibility influenced by the genetic interactions of mildew locus a6 ( Mla6) , encoding a nucleotide binding leucine-rich repeat (NLR) immu