Aging is a major risk factor for neurodegenerative diseases, yet the underlying epigenetic mechanisms remain unclear. Here, we generated a comprehensive single-nucleus cell atlas of brain aging across multiple brain regions, comprising 132,551 single-cell methylomes and 72,666 joint chromatin conformation-methylome nuclei. Integration with companion transcriptomic and chromatin accessibility data yielded a cross-modality taxonomy of 36 major cell types. We observed that transposable element (TE)
Cell-type-specific transposon demethylation and TAD remodeling in aging mouse brain
Qiurui Zeng·Joseph R. Ecker·Jasper Lee·Julia Osteen·Jessica Arzavala·Silvia Cho·Maria Luisa Amaral·Chumo Chen·Cynthia Valadon·Stella Maris Cao·Yuancheng Ryan Lu·Cindy Tatiana Báez-Becerra·William S. Owens·Zhanghao Wu·A. Klein·Jackson Willier·Ariana Barcoma·Wubin Ding·Wenliang Wang·M. Margarita Behrens·Rosa G. Castanon·Jordan Altshul·Joseph R. Nery·Wei Tian·Jonathan Rink·Bing Ren·Huaming Chen·Anna Bartlett·Nicholas D. Johnson·Hanqing Liu·Mia Kenworthy·Yuru Song·Nora Emerson·Nathan R. Zemke

