Hepatic ischemia-reperfusion injury (HIRI) is a significant contributor to liver dysfunction and failure following liver transplantation and hepatic surgical procedures. With the increasing global incidence of end-stage liver disease necessitating transplantation, strategies to mitigate HIRI have become important. Ferroptosis, a regulated form of cell death characterized by iron dependency and lipid peroxidation, has recently been recognized as a pivotal mechanism underlying hepatocellular injur