Macrophage-intrinsic and IL-9–dependent arginine metabolism promotes lung tumor growth
Anthony Cannon·Mark H Kaplan·Emily K. Bromley·Teresa L. Mastracci·Gyoyeon Hwang·Başar Bilgiçer·Abigail Pajulas·Sabrina Khan·Daniella Muallem Schwartz·Ahmed M Abdelaal·James Ropa·Cherry C L Cheung·J. Jillian Zhang·Michelle Liu Niese·Maya S. Krishnan
Tumor-associated macrophages are an abundant, tumor-infiltrating cell population that supports the evasion of tumor cells from antitumoral immune cell detection by generating an immunosuppressive tumor-immune microenvironment (TIME). The immunosuppressive function of macrophages is dictated by the cytokine environment. IL-9 is a pleiotropic cytokine that can be a positive or negative regulator of tumor growth. Our lab previously identified a protumoral role of IL-9 by expanding lung interstitial
